Diabetes II (T2D)
Type 2 diabetes (T2D) is a chronic metabolic disease characterized by persistently
high blood sugar levels caused by the body's inability to use insulin properly
(insulin resistance) and/or insufficient insulin production by the pancreas.
Low testosterone and type 2 diabetes form a bidirectional vicious cycle where each
condition worsens the other. Men with low testosterone develop increased visceral fat
and insulin resistance, raising their diabetes risk, while diabetes itself—through
mechanisms involving inflammation, insulin resistance, and obesity—further suppresses
testosterone production by disrupting the brain-testicular hormone axis and increasing
testosterone conversion to estrogen in fat tissue. This creates a self-perpetuating loop
where declining testosterone leads to more belly fat and worse blood sugar control,
which in turn causes even lower testosterone levels, making both conditions
progressively harder to treat without addressing the underlying hormonal imbalance.
Bidirectional Bandage: Type 2 Diabetes ↔ Low Testosterone
T2D → Low Testosterone Insulin resistance and hyperinsulinemia suppress
the hypothalamic-pituitary-gonadal axis. Chronic low-grade inflammation in T2D reduces
LH/FSH secretion. Increased leptin and cytokine levels inhibit testicular testosterone
production.
Low Testosterone → T2D Lower testosterone reduces muscle mass → lower
insulin sensitivity. Promotes visceral fat → worsens insulin resistance. Decreased
energy/motivation → less physical activity. Worsens glucose management and β-cell
function.
Vicious circle: T2D promotes hormonal suppression → lower testosterone
→ more fat/insulin resistance → exacerbation T2D → etc.
note for editor: links checks